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Human Growth hormone administration within normal animals leads to muscle hypertrophy, but this muscular growth is not accompanied by increased strength ( but size definitely increased)and is accompanied by alterations in protein metabolism, lipid metabolism, and the expression of other growth factors (e.g., Sirt3, MyoD, and GDF11). In contrast, there is evidence that IGF-I-induced muscle growth is not accompanied by changes in any of these growth factors. The authors conclude that, "We suggest the use of IGF-I/IGFBP-3 as an experimental target for the therapeutic manipulation of anabolic and catabolic processes within the human muscle." This is clearly a very strong finding. Although this is a well established principle in research, it will be a major challenge to evaluate. Further, there is no indication that GH supplementation of rodents or other animals leads to muscle loss or changes in other important endpoints. For this reason, the authors would caution that their findings might not translate to humans. Future studies using IGF-I-stimulated rodents would be necessary to determine whether the mechanism by which GH helps with muscle growth is similar to that seen in humans. Conclusion For the authors, the main question here is this: what is going on? If this finding were to have been true for any period that lasted 5 weeks or longer, it would seem to suggest that GH plays a dominant role in muscle growth. However, most studies that have looked at this question seem to have been of short duration, and those that did look at longer periods of time have found no consistent finding. This could relate to the small sample sizes used in some of these studies or to the fact that the mechanisms by which GH promotes muscle growth seem to vary from animal to animal. However, no conclusion is possible from a relatively small sample size. This finding is relevant to the therapeutic potential of GH. It makes it appear important but does not answer the question of whether GH is a direct therapeutic agent for anabolic and catabolic conditions such as muscle hypertrophy. We need more research to answer this question. Similar articles: